HOW IT WORKS :: bluMjk Pain Relief Science and Technology

Mode of Action

Panel A shows how bluMjk penetrates the stratum corneum or keratin (skin) via the intercellular lipid channels between the dead corneocytes. The lipophilic (definition: fat-liking, having an affinity for, tending to combine with, or capable of dissolving in lipids) component of our patented carrier system enhances the diffusion of the active ingredient, pharmaceutical grade Sodium salicylate, through the fatty intercellular lipid channels.

Panel B illustrates the layers of human skin. Below the stratum corneum and epidermis lies the dermis. The dermis is composed of cellular structures and molecules suspended in water. bluMjk’s hydrophilic (definition: having an affinity for water; readily absorbing or dissolving in water) component of the carrier system rapidly diffuses through the dermis reaching the nerves.

Panel C depicts bluMjk penetrating the cell wall of a Group C nerve fiber's axon utilizing the lipophilic component of the carrier system. Sodium salicylate is delivered to the nerve axon restoring it to its resting state and eliminating the sensation of pain.

The rapid time of onset of bluMjk's topically applied analgesic (pain killing) effect is not compatible with the reduction of pain due to the non-steroidal anti-inflammatory action of salicylate.